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Over-expression of miR-17-92 in HUVECs can protect ECs from erastin-induced ferroptosis by targeting the zinc lipoprotein (A20)-acyl-CoA synthetase long-chain family member 4 (ACSL4) axis that could protect glutathione peroxidase 4 (GPX4) and inhibit ROS production (Xiao et al., 2019)

These transporters primarily consist of solute carrier family 1 member 5 (SLC1A5, namely alanine serine and cysteine transporter system 2, ASCT2), SLC38A1 (sodium-coupled neutral amino acid transporters 1, SNAT1) and SLC38A2 (sodium-coupled neutral amino acid transporters 2, SNAT2), with ASCT2 being the most important (14, 15)